A Theory of Neuronal Malfunction in Schizophrenia
Abstract
A considerable amount of biological research into schizophrenia, mainly upon blood fluids, is performed throughout the world. The authors report their latest findings on a plasma protein factor and relate these findings to other domestic research and to recent research in other nations, particularly the Soviet Union. They hypothesize that a malcontrolled blood protein contributes to the genesis of schizophrenia by selectively altering neuronal permeability to precursors of important neurotransmitters; if this occurs, it would disrupt information processing in the brain.
Access content
To read the fulltext, please use one of the options below to sign in or purchase access.- Personal login
- Institutional Login
- Sign in via OpenAthens
- Register for access
-
Please login/register if you wish to pair your device and check access availability.
Not a subscriber?
PsychiatryOnline subscription options offer access to the DSM-5 library, books, journals, CME, and patient resources. This all-in-one virtual library provides psychiatrists and mental health professionals with key resources for diagnosis, treatment, research, and professional development.
Need more help? PsychiatryOnline Customer Service may be reached by emailing [email protected] or by calling 800-368-5777 (in the U.S.) or 703-907-7322 (outside the U.S.).