Urinary Monoamine Metabolites in Children with Minimal Brain Dysfunction
Abstract
The authors hypothesized that children with minimal brain dysfunction not secondary to neurological injury might suffer from a biochemical abnormality characterized by diminished total production of monoamines. To test this hypothesis, 24-hour urinary excretion of the principal metabolites of these substances was measured in nine children with minimal brain dysfunction and in six normal controls. No differences were found beween the groups: the hypothesis was not supported.
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