Noll chronicles how this American dementia praecox was, ironically, fertile soil for the introduction of its replacement: Eugen Bleuler’s theory of schizophrenia. Bleuler viewed hallucinations, delusions, and dementia as secondary symptoms, “which arise as reactions … to environmental influences and to [the psyche’s] own strivings” (p. 236), Splitting, another element of Bleuler’s theory, made schizophrenia attractive because “psychological processes that were split could, theoretically, be reunited.” Bleuler’s ideas of reactions to the environment, hope for cure, and a premorbid “latent” schizophrenia resonated with the evolving American concept of psychosis. A. A. Brill, a disciple of Meyer’s who had studied with Bleuler in Zurich, published the first American report of schizophrenia in the American Journal of Insanity (the precursor of the American Journal of Psychiatry) in 1909 (1). By the 1910s and 1920s, Bleuler’s schizophrenia, not yet dominant in Europe, had largely supplanted Kraeplin’s dementia praecox in America. The schizophrenia that we diagnose today is an American amalgam of these two European theories.