To the Editor: Drs. Nagy and Loveland propose that the olfactory identification deficit in Alzheimer’s disease is a result of a general semantic categorization impairment (trouble naming odors or recognizing their names) and is not due to an olfactory memory deficit. However, evidence from a number of studies has clearly shown that the deficit in olfactory identification in Alzheimer’s disease is largely the result of impairment in olfactory memory. In an elegant series of experiments, Larsson et al. (1) showed that patients with mild Alzheimer’s disease performed worse than matched healthy comparison subjects on four types of olfactory identification tasks, one of which involved the matching of specific smells to which the subject had been previously exposed without requiring him or her to name the smell. In our study of mild cognitive impairment, olfactory identification deficits accompanied by lack of awareness of these deficits predicted conversion to Alzheimer’s disease in Cox analyses (relative risk=5.91, 95% confidence interval=1.5–23.7, p<0.02), even after control for age, sex, education, and modified Mini-Mental State Examination (MMSE) and Boston Naming Test scores (data for attentional and memory measures but not the Boston Naming Test were presented in the article). This finding supports the view that the olfactory deficit in Alzheimer’s disease is not caused solely by a deficit in naming or semantic categorization. We agree with these authors’ view that olfactory evoked potentials are probably not likely to prove useful for potential clinical application in making the early diagnosis of Alzheimer’s disease.