The alphabetical organization of part 2 makes for easy reference but does not necessarily treat the content very efficiently. For instance, rather than discussing individually (and under separate headings) the often similar neurobehavioral presentations of specific central nervous system (CNS) processes such as abscess, tumor, stroke, and traumatic brain injury, a more focused approach involving an examination of the neurobehavioral syndromes (aphasia, agnosia, neglect, apathy, abulia, disinhibition, and personality change) caused by disruption of specific parts of the brain, regardless of etiology, would likely be more efficient and informative. Other subjects in this section could be treated in greater depth, including seizures, Parkinson’s disease, and pseudobulbar affect. In the case of seizures, a broader conceptual discussion of ictal, postictal, and interictal states and their relationships to behavior would be helpful. For Parkinson’s disease, further discussion of psychotic symptoms, their relationship to dopaminergic treatments, the use of atypical antipsychotic agents in relation to extrapyramidal symptoms, and a broader elaboration of parkinsonism as a syndrome would benefit the reader. As for pseudobulbar affect, which is discussed with multiple sclerosis, further discussion could have focused on the important differentiation and occasional disconnection between objective findings and subjective mood state in the evaluation of patients with mental symptoms and neurological disorders.