Our studies concerned with the clinical effects of the administration of taraxein, a protein isolated from the serum of schizophrenic patients and not obtained from normal serum through our processing procedure, are summarized. In addition, we reported studies which suggest that this substance apparently is present in whole serum and is not a product activated through our processing procedure. One study concerned with the attempt to isolate taraxein following the administration of D-LSD-25 and a study describing the isolation of taraxein from patients receiving Thorazine were described. These experiments suggested that the LSD psychoses do not result from the formation of taraxein in nonpsychotic volunteers and that Thorazine does not produce clinical improvement by destroying or reducing levels of taraxein.Abstract Teaser